Looks like youve clipped this slide to already. Tap here to review the details. The air is therefore trapped in the lungs, making it hard for the lungs to send the right amount of oxygen to the rest of the body (McCance & Huether, 2019). Nurses care for patients with COPD across the spectrum of care, from outpatient to home care to emergency department, critical care, and hospice settings. Because this study aimed to explore nurse observations of COPD patients in a naturalistic setting (during home visits), eligible patients were identified and included as a third party during the data collection process. You can read more about the PCG tracks for patients and caregivers and for health care professionals. COPD - Physical Findings. Art of Assertive Commnunication, how to be visible in social media by Shreed UiPath Automation Cloud Robots - Best Practises session 2.pptx, How To Restore Tree From Ancestry To Family Tree Maker, INVERTING BUCK-BOOST DCDC CONVERTER DESIGN CHALLENGES. At the end of the end of the 16th International Family Nursing Conference, attendees should be able to discuss global advancements in family nursing education for students and professionals. We've updated our privacy policy. Copyright 2023 Freepik Company S.L. Click here to review the details. How to Add, Duplicate, Move, Delete or Hide Slides in Google Slides, How to Change the Slide Size in Google Slides, 26 different slides to impress your audience, Contains easy-to-edit graphics such as graphs, maps, tables, timelines and mockups, Includes 500+ icons and Flaticons extension for customizing your slides, Designed to be used in Google Slides and Microsoft PowerPoint, 16:9 widescreen format suitable for all types of screens, Includes information about fonts, colors, and credits of the free resources used. copd. p.1314-1318. management of stable copd shyam rao may 2014. objectives . Enjoy access to millions of ebooks, audiobooks, magazines, and more from Scribd. chronic obstructive pulmonary disease. Youll also find a list of resources for more help managing your COPD. A focused respiratory system assessment includes collecting subjective data about the patient's history of smoking, collecting the patient's and patient's family's history of pulmonary disease, and asking the patient about any signs and symptoms of pulmonary disease, such as cough and shortness of breath. The Basics of COPD contains helpful information for individuals new to COPD. The diagnosis is confirmed by spirometry.Other tests may include a 6-minute walk test, laboratory testing, and radiographic imaging.. Assessment - A diagnosis of COPD should be considered in patients over the age of 35 who have a risk factor (generally smoking) and who present with exertional breathlessness, chronic . 4. Clipping is a handy way to collect important slides you want to go back to later. 21 slides. Center of disease control and prevention. Background: Qualitative research on long COVID by subtype has not yet occurred. PPT layouts enhanced with lung - senior woman gets first aid background and a . COPD Nursing Diagnosis and Care Plan. Energy-conserving tips are presented. Do not sell or share my personal information, 1. You will also learn useful health and safety tips for managing your COPD. or chronic bronchitis that was dr bruce davies. COPD can lead to increased vulnerability by exclusion from participating fully in society. Pneumothorax occurs when the parietal or visceral pleura is breached and the pleural . the airway and out of the lungs permanent and Scavenger Hunt Week 3, Choice 1 Here finally is your chance to see The many facets of creativity; Theories, presentations, lessons & such Without reality are nothing much. Sputum gram stain and culture (for COLD exacerbation), Imaging Chest radiography Emphysema: obvious bullae, paucity of parenchymal markings, or hyperlucency Hyperinflation: increased lung volumes, flattening of diaphragm Does not indicate chronicity of changes Chest CT Definitive test for establishing the diagnosis of emphysema, but not necessary to make the diagnosis, Diagnostic Procedures Pulmonary function tests/spirometry Chronically reduced ratio of FEV1 to forced vital capacity (FVC) In contrast to asthma, the reduced FEV1 in COLD seldom shows large responses (>30%) to inhaled bronchodilators, although improvements up to 15% are common. Hallmark symptom - Dyspnea Chronic productive cough Minor hemoptysis pink puffer blue bloater. Widescreen (16:9) Presentation Templates. Provided by: Wad5. Presentations will focus on current and critical issues to provide oncology nurses with practical information that can be implemented in the practice setting. Typically performed by isoelectric focusing of serum, which reflects the genotype at the PI locus for the common alleles and many of the rare PI alleles Molecular genotyping can be performed for the common PI alleles (M, S, and Z). Most patients have elements of each. As pulmonary sequelae constitute a serious long COVID subtype, exploring patient experience and needs can generate knowledge to guide nursing practice. CHANGE IN COLOUR OF SPUTUM . It appears that you have an ad-blocker running. Join Actively Recruiting Research Studies, Bronchiectasis and NTM Research Registry (BRR), COPD Patient Powered Research Network (COPD PPRN), Patient-Inspired Validation of Outcome Tools (PIVOT), COPD Biomarker Qualification Consortium (CBQC), COPD Assessment Test (CAT) / Chronic Airways Assessment Test (CAAT), Medical and Scientific Advisory Committee.
Operating Room Nurse/ Clinical Research Nurse . Often with COPD, patients you will see some combination of both presentations seen in chronic bronchitis and emphysema. Chronic obstructive pulmonary disease (copd) by aseem, Chronic obstructive pulmonary disease (copd), Obstructive And Inflammatory Lung Disease. -Continuous evaluation of classes and the course to . "Blue bloaters" are patients with predominant bronchitiscyanosis and edema. Chronic Obstructive Pulmonary Disease . Chronic obstructive pulmonary diseases & Nursing care. View COPD Powerpoint.pptx from NUR 395 at Ameritech College of Healthcare, Draper. dinner was great, coffee is brewing and you are all debating, COPD - . progressive syndrome expiratory airflow obstruction chronic airway and lung parenchyma inflammation. Many in this series of 12- to 20 . Instant access to millions of ebooks, audiobooks, magazines, podcasts and more. 3. Nursing management of chronic obstructive pulmonary disease. 28-7, Emphysema Pathophysiology
Hyperinflation of alveoli
Destruction of alveolar walls
Destruction of alveolar capillary walls
Narrowed airways
Loss of lung elasticity, Emphysema Pathophysiology Two types:
Centrilobular (central part of lobule)
Most common
Panlobular (destruction of whole lobule)
Usually associated with AAT deficiency, Emphysema Pathophysiology Structural changes are:
Hyperinflation of alveoli
Destruction of alveolar capillary walls
Narrowed, tortuous small airways
Loss of lung elasticity, Emphysema Pathophysiology Small bronchioles become obstructed as a result of
Mucus
Smooth muscle spasm
Inflammatory process
Collapse of bronchiolar walls
Recurrent infections production/stimulation of neutrophils and macrophages release proteolytic enzymes alveolar destruction
inflammation, exudate, and edema, Emphysema Pathophysiology Elastin and collagen are destroyed
Air goes into the lungs but is unable to come out on its own and remains in the lung
Causes bronchioles to collapse, Emphysema Pathophysiology Trapped air hyperinflation and overdistention
As more alveoli coalesce, blebs and bullae may develop
Destruction of alveolar walls and capillaries reduced surface area for O2 diffusion
Compensation is done by increasing respiratory rate to increase alveolar ventilation
Hypoxemia usually develops late in disease, Emphysema Clinical Manifestations
Dyspnea
Progresses in severity
Patient will first complain of dyspnea on exertion and progress to interfering with ADLs and rest, Emphysema Clinical Manifestations Minimal coughing with no to small amounts of sputum
Overdistention of alveoli causes diaphragm to flatten and AP diameter to increase, Emphysema Clinical Manifestations
Patient becomes chest breather, relying on accessory muscles
Ribs become fixed in inspiratory position, Emphysema Clinical Manifestations Patient is underweight (despite adequate calorie intake), Chronic Bronchitis Pathophysiology Pathologic lung changes are:
Hyperplasia of mucus-secreting glands
in trachea and bronchi
Increase in goblet cells
Disappearance of cilia
Chronic inflammatory changes and narrrowing of small airways
Altered fxn of alveolar macrophages infections, Chronic Bronchitis Pathophysiology Chronic inflammation
Primary pathologic mechanism causing changes
Narrow airway lumen and reduced airflow d/t
hyperplasia of mucus glands
Inflammatory swelling
Excess, thick mucus, Chronic Bronchitis Pathophysiology
Greater resistance to airflow increases work of breathing
Hypoxemia and hypercapnia develop more frequently in chronic bronchitis than emphysema, Chronic Bronchitis Pathophysiology Bronchioles are clogged with mucus and pose a physical barrier to ventilation
Hypoxemia and hypercapnia d/t lack of ventilation and O2 diffusion
Tendency to hypoventilate and retain CO2
Frequently patients require O2 both at rest and during exercise, Chronic Bronchitis Pathophysiology Cough is often ineffective to remove secretions because the person cannot breathe deeply enough to cause air flow distal to the secretions
Bronchospasm frequently develops
More common with history of smoking or asthma, Chronic Bronchitis Clinical Manifestations Earliest symptoms:
Frequent, productive cough during winter
Frequent respiratory infections, Chronic Bronchitis Clinical Manifestations Bronchospasm at end of paroxysms of coughing
Cough
Dyspnea on exertion
History of smoking
Normal weight or heavyset
Ruddy (bluish-red) appearance d/t
polycythemia (increased Hgb d/t chronic hypoxemia))
cyanosis, Chronic Bronchitis Clinical Manifestations Hypoxemia and hypercapnia
Results from hypoventilation and airway resistance + problems with alveolar gas exchange, COPD Complications Pulmonary hypertension (pulmonary vessel constriction d/t alveolar hypoxia & acidosis)
Cor pulmonale (Rt heart hypertrophy + RV failure)
Pneumonia
Acute Respiratory Failure, COPD Diagnostic Studies Chest x-rays early in the disease may not show abnormalities
History and physical exam
Pulmonary function studies
reduced FEV1/FVC and residual volume and total lung capacity, COPD Diagnostic Studies ABGs
PaO2
PaCO2 (especially in chronic bronchitis)
pH (especially in chronic bronchitis)
Bicarbonate level found in late stages COPD, COPD Collaborative Care Smoking cessation
Most significant factor in slowing the progression of the disease, COPD Collaborative Care: Drug Therapy
Bronchodilators as maintenance therapy
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